Figure 6
From: Experimental obstructive cholestasis: the wound-like inflammatory liver response
Ischemia-revascularization and leukocytic phenotypes during the evolution of obstructive cholestasis. Predominance of oxidative stress with edema and enzymatic stress with infiltration by leukocytes and Kupffer cell activation. AC: arterial capillar; B: bacteria; C: cholangiocyte; ECM: extracellular matrix; HSC: hepatic stellate cell; KC: Kupffer cell; LPS: lipopolysaccharide; MC: mast cell; N: Neutrophyl; PM: portal myofibroblast; PP: peribiliary arterial plexus; RBC: red blood cells; SC: stem cells; SS: sinusoidal space; Th1: T cell h1; Thi: intraepithelial lymphocyte.